I have blood again after one week of my period.

There is blood again a week after the menstrual period. After a week of menstruation, there is bleeding, not too much, or candidal vaginitis cannot be ruled out, but it is best to do a re-examination. For vaginitis, you can take vaginal suppositories combined with oral anti-inflammatory drugs, wash with potassium permanganate daily, dilute it, and then add suppositories to treat anti-inflammatory. Pay strict attention to health care when changing underwear, do not eat raw, cold or irritating food, do not have unclean sex life, pay attention to the quality of sex life and do not have it too frequently, and try to take protective measures such as condoms.

1. Damage to the vessel wall is common in hypoxia, which causes degeneration of capillary endothelial cells; sepsis (especially meningococcal sepsis), rickettsial infection, hemorrhagic fever, snake venom, organophosphorus poisoning, etc. cause damage to the capillary wall; some drugs can cause allergic vasculitis; vitamin C deficiency can cause rupture of the capillary basement membrane, decrease in pericapillary collagen, and separation of endothelial cell junctions, resulting in increased permeability of the vessel wall; in Henoch-Schönlein purpura, allergic vasculitis is caused by the deposition of immune complexes on the vessel wall.

2. Thrombocytopenia and dysfunction The normal number and quality of platelets are important factors in maintaining normal capillary permeability. When the number of platelets decreases to a certain level, exudative bleeding may occur. For example, aplastic anemia, leukemia, and extensive tumor metastasis in the bone marrow can reduce platelet production; idiopathic thrombocytopenic purpura, thrombotic thrombocytopenic purpura, and DIC cause excessive platelet destruction or consumption; certain drugs induce an immune response to antigen-antibody complexes in the body, and the immune complexes formed are adsorbed on the surface of platelets, causing the latter to be phagocytosed by macrophages along with the immune complexes; some bacterial endotoxins and exotoxins also have the effect of destroying platelets.

Structural and functional defects of platelets can also cause exudative bleeding. Many of these diseases are congenital, such as thrombasthenia (lack of fibrin receptors on the platelet cell membrane) and storage pool disease (lack of one or more granules, resulting in insufficient ADP reserves; it can also occur due to acquired damage to bone marrow megakaryocytes), in which the platelet adhesion ability is defective; in Bernard-Soulier syndrome (lack of von Willebrand factor receptors on the platelet cell membrane), platelets cannot adhere to collagen fibers, which can lead to coagulation disorders or bleeding tendencies.