What are the symptoms of caesarean section blood clots

The probability of women dying during childbirth in ancient times was much higher than it is now. Because there was no help from medical technology, they could only give birth on their own, and no caesarean section was available, no matter how difficult it was. Nowadays, if there are problems with natural childbirth, doctors will promptly choose to use caesarean section to help the baby be born. This not only reduces the pain of the new mother but is also safer. So what are the symptoms of blood clots during cesarean section?

Generally, venous thrombosis is relatively rare after childbirth. Its occurrence may be due to the fact that the mother's blood is in a hypercoagulable state after childbirth, coupled with postpartum fatigue and weakness, and incision pain that leads to more bed rest, which slows the blood circulation in the veins of the lower limbs and makes blood easily accumulate in the veins.

A thrombus is a small piece of blood formed when blood flows onto the surface of a peeling or repaired part of the inner surface of a blood vessel in the cardiovascular system. In variable flow dependent patterns, thrombi are composed of insoluble fibrin, deposited platelets, accumulated leukocytes, and entrapped erythrocytes.

Thrombosis is a multifactorial process in which a group of genetic and environmental factors interact and influence each other. The main characteristics of common thrombosis patients in clinical practice are familial inheritance, recurrent attacks, severe symptoms, abnormal sites of thrombosis formation, and young onset of disease.

Suspicion of arterial or venous thrombosis or thromboembolism requires objective evidence. Angiography is the standard of care, but ultrasonography, performed with skill, can also examine superficial vessels and the heart.

Among patients with spontaneous deep vein thrombosis documented by venography, a genetic predisposition is present in 25% to 50% of cases. The presence of a congenital defect in the anticoagulant mechanism (eg, factor V resistance to activated protein C, homocystinemia, protein C deficiency, protein S deficiency, antithrombin deficiency, fibrinolytic insufficiency) is sufficient to cause venous thromboembolism when combined with a thrombotic stimulus (eg, surgery, pregnancy, contraceptive use, antiphospholipid antibodies). Patients with multiple thrombosis in the early stage have a significantly higher frequency and severity of thrombosis than those with a single thrombosis.

Antithrombotic therapy is the use of thrombolytic drugs, antiplatelet drugs and anticoagulants. When developing an antithrombotic treatment strategy, the first thing to note is

Thrombolytic drug therapy, because thrombolytic drugs can remove an already formed clot. Antithrombotic therapy is varied, depending on whether the affected part is the venous or arterial circulation system; the extent and location of vascular involvement; the risk of thrombosis extension, embolism or recurrence; and the relative benefits and harms of antithrombotic therapy and bleeding.