Causes of free thyroxine in pregnant women

In today's life, many pregnant women often experience miscarriage, premature birth, or even fetal cessation of development. These are caused by not paying attention to our own body's condition in normal times. For example, the common free thyroxine in pregnant women controls the thyroid gland of pregnant women. If there is a problem, it is likely to affect the slow growth of the fetus.

Generally speaking, patients with mild hyperthyroidism and those whose condition can be well controlled after treatment can become pregnant, and most of them can achieve good pregnancy results under the supervision of obstetricians and internal medicine physicians. Patients with severe hyperthyroidism or those whose condition is difficult to control have more complications for both the mother and the fetus after becoming pregnant. There is a long-acting thyroid stimulating hormone in the blood of patients with hyperthyroidism, which promotes thyroid function. This substance can enter the fetal blood circulation through the placenta and cause temporary hyperthyroidism in the fetus. The fetus can absorb iodine and synthesize thyroxine at 15 weeks of pregnancy. However, after the mother becomes pregnant, the kidney's reabsorption of iodine decreases, which can easily lead to iodine deficiency, causing the fetus to absorb less iodine and causing changes in thyroid function. During the treatment of pregnant women, if too much iodine is given, it can also be absorbed by the fetus through the placenta. Therefore, the child may develop hypothyroidism or hyperthyroidism after birth. In summary, the drugs taken by the mother and pathological changes may affect the fetus. In severe cases, it can cause miscarriage, premature birth, intrauterine growth retardation, and even neonatal asphyxia.

Both pregnancy and hyperthyroidism can increase the cardiac burden of pregnant women and induce heart failure in patients with severe hyperthyroidism. Patients with this disease are also many times more likely to have pregnancy-induced hypertension than those without hyperthyroidism. Due to the reduction in energy storage, uterine contraction weakness is likely to occur, which may prolong labor and increase postpartum bleeding. At the same time, the chance of postpartum infection will increase. In addition, the thyroid gland may increase slightly in size and thyroxine may increase slightly after pregnancy. It is particularly worth mentioning that for severely ill hyperthyroid patients who must undergo surgical delivery, both anesthesia and surgery may induce hyperthyroid crisis. Of course, whether a patient with hyperthyroidism can become pregnant can only be decided by the doctor who usually treats her after a detailed physical examination. If the condition is mild, you can get pregnant, but you must receive medical treatment to control the condition before pregnancy, have regular prenatal check-ups after pregnancy, and receive treatment under the guidance of a doctor to minimize the occurrence of maternal and fetal complications.

In the article, we learned about the free thyroxine in pregnant women. If there is an abnormality, which we often call hyperthyroidism, goiter, fatigue, and lack of strength can cause delayed uterine development.