Endometrial hyperplasia is

Uterine wall hyperplasia can generally be divided into several categories: one is simple uterine wall hyperplasia; one is multiple uterine wall hyperplasia; and the other is endometrial cancer. Endometrial cancer is mainly caused by the excessive proliferation of the uterine wall's own cells, which develops into a condition with more genetically modified cells. It usually needs to be treated properly to avoid delaying the disease. If the uterine wall does not undergo extensive hyperplasia, it is mainly caused by polycystic ovaries, tumors on the uterus and ovaries, or menstrual metabolic disorders.

It is necessary to clarify how to carry out treatment based on the actual reasons. Simple hyperplasia of the uterine wall should be caused by a simple increase in estrogen, mainly due to mental anxiety or disordered hormone metabolism, all of which lead to the continuous thickening of the functional layer of the uterine wall. Therefore, reliable treatment should be carried out according to the cause of the disease.

The normal uterine wall changes regularly. It is divided into the proliferative phase, metabolic phase, and physiological phase. Uterine wall hyperplasia refers to the long-term and continuous stimulation of estrogen by the uterine wall, without ovulation, estrogen resistance, and regular metabolic changes, but in a proliferative stage for a long time. It can also be caused by inflammation, infection, or drug irritation. It can be divided into simple hyperplasia, complex hyperplasia, and intestinal metaplasia. Symptoms such as abdominal pain and non-menstrual bleeding in the vagina may occur. Endometrial hyperplasia has a pathological tendency, but it also has cross-talk.

The symptoms of uterine wall hyperplasia are first manifested as irregular menstruation, sometimes long-term amenorrhea, and sometimes irregular vaginal bleeding. B-ultrasound will show endometrial thickening, generally more than 15mm. The main reason is anovulation. Although the egg grows, it is not discharged. Without ovulation, there is no estrogen production. Simple uterine development will produce estrogen. Under the action of a single estrogen, the uterine wall will have excessive hyperplasia. Over time, the uterine wall will fall off and bleed. Curettage and ablation are required for pathological examination, and the next step of treatment is determined based on the pathological results. If pathological results show endometrial hyperplasia, estrogen treatment is necessary.