The growth of the uterine wall actually changes with the changes in the body's growth hormones. The growth of the uterine wall is mainly the effect of estrogen and estrogens. The function of estrogen is to cause the endometrium to thicken and proliferate excessively, and even cause uterine intrauterine polyp-like hyperplasia, and even intestinal metaplasia of the uterine wall and endometrial cancer. This is the function of estrogen. The effect of estrogen is to make the uterine wall thicker and turn it into the metabolic uterine wall. After it turns into the metabolic uterine wall, the uterine wall can fall off and menstruation can occur. This is the effect of estrogen on the uterine wall. Small amounts of estrogen can actually counteract the effects of small amounts of estrogen on the uterine wall, so estrogen can sometimes soften the uterine wall. The hormones that act on the uterine wall are mainly estrogen and estrogens. Among them, the hormone that causes the uterine wall to proliferate is estrogen, and the defensive hormone is estrogen. The growth hormone that can promote endometrial hyperplasia is estrogen. The uterine wall is the target organ of estrogen, and both estrogen and progesterone have a buffering effect on the uterine wall. Estrogen mainly stimulates the growth of the uterine wall and promotes egg maturation and ovulation. Estrogen promotes the metabolism of the uterine wall after ovulation, and before the menarche, the levels of estrogen and progesterone decrease at the same time. The uterine wall loses hormone support and normal menstruation occurs. However, if the estrogen level is too high, it will cause excessive proliferation of the uterine wall. There are conditions such as increased menstrual flow or abnormal uterine bleeding due to imbalanced function. Therefore, only when the ratio of estrogen and progesterone is normal can a normal menstrual period be maintained. The growth hormones that promote endometrial hyperplasia are female hormones, such as estradiol, estriol, and estrone. The higher the estrogen level, the more obvious the endometrial hyperplasia. Estrogen levels also change throughout the physiological cycle. For example, before ovulation and before menarche, estrogen levels rise relatively, so the thickness of the endometrium also changes. Normal estrogen maintains various female characteristics, but if the estrogen level is too high, it can cause uterine wall disease and even develop into endometrial cancer. |
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