Can the thickness of the inner membrane recover on its own?

Can the thickness of the inner membrane recover on its own?

Thick endometrium is mainly caused by inflammation of the endometrium or endocrine disorders in the body. As a common gynecological disease, endometritis is actually not well known by many female friends. The increase in endometrial thickness can be detected through B-ultrasound examination, six sex hormone tests, diagnostic curettage and scraping, etc.

The thickening of the endometrium is mainly caused by inflammation of the endometrium, or endocrine disorders, drug stimulation and other factors, which lead to excessive growth of the endometrium. Usually, a large amount of hormones can be used to change the hormone levels in the body to achieve normal endocrine conditions and treat the disease. In addition, you can also use progesterone and other drugs to regulate according to the doctor's advice. This method of adjustment is relatively slow and requires long-term adjustment. Endometrial thickening can recover on its own, which is related to the fluctuation of hormone levels in the body, so the possibility of recovery on its own exists, but the probability is relatively low.

Examinations for endometrial thickening include B-ultrasound examination, six sex hormone tests, diagnostic curettage and scraping, hysteroscopy, etc.

1. Hysteroscopic examination: Use hysteroscopy to observe the appearance of the endometrium and see the condition of the endometrium, and perform curettage or negative pressure aspiration under direct vision.

2. Serum hormone measurement: Use B-ultrasound or laparoscopy to determine whether polycystic ovary is present.

3. X-ray or CT examination: Examination of the pituitary sella turcica and fundus visual field to rule out pituitary tumors.

4. Basal body temperature measurement: It can be used to understand whether ovulation has occurred/whether the function of the corpus luteum is healthy based on the arc of the body temperature rise and the length of time the rise is maintained.

Atypical endometrial hyperplasia must be differentiated from the other two types of simple hyperplasia and complex hyperplasia. At the same time, attention should be paid to distinguishing it from early endometrial adenocarcinoma.

How to treat endometrial thickening

The treatment of endometrial thickening should be symptomatic according to different causes. First, check whether the patient has polycystic ovary, functional ovarian tumors or other diseases caused by endocrine dysfunction, and then perform targeted treatment. Drug treatment and surgery are generally used for treatment.

1. Ovulation induction therapy: This treatment method is mostly used for young patients. Patients should first measure their basal body temperature. If they are confirmed to be monophasic anovulatory, this treatment method can be used. The ovulation-inducing drugs include clomiphene and chorionic gonadotropin. The dosage of clomiphene is 50-100 mg, once a day, taken on the 5th to 9th day of the menstrual cycle. If necessary, the medication period can be extended by 2-3 days.

2. Curettage and dilation for women in the reproductive period: Generally, one curettage can control bleeding. If bleeding still occurs after curettage, hysteroscopy and B-ultrasound should be performed to rule out submucosal myoma or other organic lesions. Women in the reproductive period may also be infertile and have clinical manifestations of anovulation due to polycystic ovary syndrome, which should be treated according to polycystic ovary syndrome.

3. Menopausal transition period: It is often anovulatory functional uterine bleeding. If menstruation is infrequent and the amount of blood is heavy or the bleeding time is long after curettage and hemostasis, progesterone treatment should be given every two months, and follow-up observation should be conducted after 3 cycles.

4. Late menopause: You should ask whether to use estrogen replacement therapy alone. After curettage, the replacement therapy can be suspended or progesterone can be added. Progestogens can inhibit the proliferation of the endometrium caused by estrogen.

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