Uterine endometriosis cysts are an uncommon tumor that often occurs at traumatic sites. Surgical intervention for Bartholin's gland cysts or cysts usually provides a good environment for the occurrence of this disease. Uterine ectopic cysts 1. Brief description: The main manifestations of this disease vary depending on the location, but most patients have local symptoms of the private parts, that is, local swelling and pain. 2. Causes of disease: 1. Endometrial implantation: The shed uterine wall still has a certain capacity. Whether it is during menstruation or surgery, the endometrial fragments that fall off are likely to migrate and grow in new locations. For example, factors such as hymen occlusion, cervical stenosis, and retroverted uterus can hinder the flow of menstruation, and menstruation can flow back into the pelvis through the bilateral fallopian tubes. In addition, if the negative pressure in the uterine cavity is suddenly reduced during abortion curettage, uterine examination during menstruation can also indicate uterine wall transplantation. During cesarean section or uterine removal, if part of the endometrial tissue is accidentally lost into the abdomen, endometriosis may also occur. 2. Immediate spread: Endometriosis of the myometrium may spread directly from the cervix. 3. Tissue biochemistry: The germinal epithelial cells of the pelvic retroperitoneum or uterus and ovaries and the uterine wall are all formed by the division of renal tubular epithelial cells. In response to inflammation or other irritants, retroperitoneal or utero-ovarian ablative germinal epithelial cells may form the uterine wall. 4. Venous or lymphatic metastasis: The endometrium may migrate to distant locations outside the uterus via veins or lymphatic vessels, causing endometriosis in the lungs, bladder, intestines, and other locations. Uterine ectopic cysts III. Injury: If uterine endometriosis is not treated in time, the uterine wall will continue to develop and spread, and will gradually cause uterine wall cysts in the ovaries, fallopian tubes and pelvis, causing adhesions between the fallopian tubes and surrounding tissues. Uterine ovarian adhesions or uterine endometriosis cysts destroy normal uterine ovarian tissue and hinder ovulation. Pelvic adhesions lead to protection of the ovaries and the fimbria of the fallopian tubes, and the egg cells cannot enter the fallopian tubes. If the egg cell cannot enter, fertilization cannot be carried out, resulting in infertility. Director Zhang Yukun also said that due to the occurrence of uterine ectopic disease, the sperm and egg cannot reach the uterus normally, but accumulate and develop in the fallopian tubes, resulting in ectopic pregnancy. Ectopic pregnancy can cause internal bleeding in women and threaten their lives. Therefore, we should actively prevent endometriosis and treat it as soon as possible. Uterine ectopic cysts IV. Pathogenesis: 1. The pathogenesis is not completely clear, and the core basic theory is Sampson's menstrual retrograde implantation, renal tubular metaplasia and its induction theory. 2. The uterine wall needs to go through the process of adhesion, erosion and angiogenesis inside and outside the uterine cavity. When the disease occurs after implantation and development, the characteristics of the current endometrium may play a decisive role. 3. During the above process of endometrial disruption, the body's systemic and local immune status and functions, growth hormones, cytokines and enzymes all play a key role. 4. Endometriosis tends to cluster in large families. 5. External air pollution may have a certain impact. |
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