Lidocaine can affect pregnancy

Lidocaine is a local anesthetic and antiarrhythmic drug. It is a compound of cocaine, but it does not have the hallucinations and addictive properties of cocaine. Lidocaine hydrochloride is a milky white crystalline powder, soluble in water, and has similar toxicity to procaine, but its local anesthetic effect is extremely strong and long-lasting, with good surface penetration, and can be injected or used as a topical anesthesia. So, will using lidocaine affect pregnancy?

Does lidocaine affect pregnancy?

Any medications taken orally within 30 days of pregnancy have no effect on the fetus. That is to say, the gestational sac has not yet reached the uterine cavity. There is no blood respiratory system established. It is not easy to be affected. Nowadays, if you want to have a baby, you may go to the hospital to complete various prenatal checkups to ensure that everything goes well during the pregnancy.

Lidocaine is a good partial local anesthetic that generally takes effect one to three minutes after application and lasts for one to three hours. Used to treat oral ulcers. It is no longer commonly used as a heart rhythm disorder drug because many people are concerned that it may have long-term side effects. A few people have skin allergies to lidocaine.

It was used in 1963 to treat arrhythmias. It is currently a drug for preventing acute myocardial infarction and various heart diseases with high complications of rapid ventricular arrhythmias. It is the preferred drug for ventricular premature beats, ventricular tachycardia and ventricular tremors in acute myocardial infarction.

Lidocaine is a fluorophenyl local anesthetic. After absorption from the blood or intravenous administration, it has significant excitatory and inhibitory biphasic effects on the central nervous system, and there is no precursor excitation. When the blood concentration is low, analgesia and drowsiness occur, and the pain threshold increases; with the increase of dosage, the efficacy or side effects increase, and it has anticonvulsant effect at sub-toxic blood concentrations; when the blood concentration exceeds 5 mg·ml-1, convulsions may occur. At low doses, this product can promote the loss of K in the body, reduce the heart's self-control ability, and have an anti-ventricular arrhythmia effect; at therapeutic doses, it has no significant effect on the body's electrical activity, atrioventricular conduction and cardiac contraction; further increases in blood drug concentrations can cause a slowing of cardiac conduction rate, atrioventricular conduction block, inhibition of cardiac contraction and a decrease in cardiac output.