Hyperplastic endometrium

Hyperplastic endometrium

Endometrial hyperplasia is a relatively common gynecological disease. Of course, this kind of health problem is more serious for women. If the endometrial hyperplasia is benign, it is likely to develop into benign uterine fibroids. If it is malignant, it can easily deteriorate into uterine cancer. No matter which disease it is, it will bring great pain to women and needs timely treatment!

1. Normal thickness of the endometrium

1. Proliferative phase: The proliferative phase of women is generally from the fifth to the ninth day of menstruation. At this time, the endometrium of women is very thin. In the early stage of endometrial hyperplasia, the thickness of the endometrium is about 4-6mm. When it reaches the middle of the proliferative area, the thickness of the endometrium will increase to 8-10mm. At the end of the proliferative phase, the thickness of the endometrium will increase to about 9-10mm.

2. Secretory phase: Generally, within 1-5 days after female ovulation, the endometrium of female friends will continue to thicken. Within 6-10 days after ovulation, the endometrial secretion of female friends will increase significantly. In the late secretory phase, the endometrium of female friends is generally about 5-6mm.

3. Menstrual period: Within 1-4 days of women's menstruation, the hormone level in women's bodies will decrease, and the women's endometrium will shed to a certain extent. The shed necrotic cells will be discharged with the menstrual blood. At this time, the thickness of women's endometrium is about 8-10mm.

2. Causes of Endometrial Hyperplasia

1. The body is stimulated by high levels of estrogen, either endogenously (such as endocrine functional tumors in the ovary or pituitary gland) or exogenously (such as estrogen replacement therapy).

2. Due to the lack of ovulation, the endometrium is in a state of proliferative growth for a long time and lacks periodic secretory phase transformation.

3. Simple hyperplasia is considered to be a physiological response of the endometrium to the body's high estrogen state. The most common cause is anovulatory menstruation, which often occurs in women before menarche or menopause.

3. Principles of treatment for endometrial hyperplasia

The treatment of atypical endometrial hyperplasia must first determine the diagnosis and the cause of the atypical hyperplasia, including whether there is polycystic ovary, functional ovarian tumors or other endocrine dysfunction. Those with any of the above conditions should receive targeted treatment. At the same time, symptomatic treatment can be started for atypical endometrial hyperplasia, using drug therapy or surgical treatment. The choice of these two treatment options should be based on age, type of endometrial hyperplasia, fertility requirements, etc. It can be appropriately combined with some traditional Chinese medicines for preventing cancer. Scientific evidence has shown that ginsenoside rh2 has the effect of inhibiting the spread of cancer cells and inducing apoptosis of cancer cells.

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