Many people will experience symptoms of angina pectoris due to excessive fatigue or long-term insomnia, and may even experience temporary chest tightness and shortness of breath. This kind of situation is more likely to occur among female friends. However, this type of angina pectoris is generally a physiological phenomenon and not a real lesion. Below we will give you a detailed analysis of the causes of angina pectoris, and also introduce to you the symptoms of angina pectoris in women. Most cases of chest tightness and pain in women are not heart problemsFor a normally healthy young woman, these phenomena are generally manifestations of functional changes in the patient's organs, and are rarely manifestations of organic diseases. In other words, it is generally not caused by the phenomenon of "myocardial ischemia". The so-called myocardial ischemia is mostly caused by pathological stenosis of the coronary arteries. One of the most common reasons is arteriosclerosis, and arteriosclerosis is mostly prone to occur in middle-aged and elderly people. If young people, especially women, experience chest tightness, chest pain or even shortness of breath, it is likely that cardiac neurosis is at work. Only very few are caused by coronary heart disease, coronary artery inflammation, coronary artery aneurysm, myocardial bridge, myocarditis, and congenital coronary artery malformation.Cardiac neurosis is mostly caused by factors such as anxiety, tension, emotional excitement, mental trauma, overwork, too little physical activity, and lack of proper exercise of the circulatory system, which results in excessive cardiovascular reaction and this disease after the patient has a little activity or a little fatigue.
Angina pectoris refers to a group of syndromes with precordial pain as the main clinical manifestation caused by insufficient coronary blood supply due to atherosclerotic stenosis, temporary myocardial ischemia and hypoxia. Angina pectoris occurs when there is a conflict between the blood supply of the coronary arteries and the blood demand of the myocardium, and the coronary blood flow cannot meet the metabolic needs of the myocardium, causing acute and temporary ischemia and hypoxia of the myocardium. When coronary arteries develop atherosclerotic lesions or suffer from coronary artery spasm, coronary artery stenosis or blockage due to other reasons, coronary blood flow decreases and can only supply myocardial metabolism when the patient is at rest or doing light activity. Therefore, myocardial ischemia and hypoxia generally do not occur in a resting state. However, when the cardiac load increases, such as when tired, emotionally excited, exerting oneself, cold or having a full meal, the myocardial oxygen demand increases, but the coronary arteries cannot dilate compensatorily as normal, and may even react abnormally and spasm, causing temporary myocardial ischemia and angina pectoris. |
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