Many pregnant women will experience an increase in D2 aggregates in their bodies during pregnancy, but the increase in D2 aggregates generally has no direct correlation with fetal arrest. In other words, although many pregnant women have an increase in D2 aggregates, it generally does not directly affect the growth and development of the fetus itself. However, if the D2 aggregate increases significantly, it may cause the pregnant woman's blood to be in a hypercoagulable state, which may lead to venous thrombosis and other conditions. If there is a significant increase in D2 aggregates, the blood condition of the pregnant woman should be closely monitored to avoid causing greater impact on her life and health. In addition, we also need to monitor the condition of the fetus in the belly and the growth and development of the fetus itself. However, under normal circumstances, a slight increase in D2 aggregates will not have a big impact on fetal arrest. If the increase level is relatively high, it also depends on the actual life standard of the pregnant woman. But in general, under normal circumstances, the increase of D2 aggregates has no direct effect on fetal arrest. D-dimer mainly reflects the dissolution of free fatty acids. The elevated or positive type is common in cases where the secondary free fatty acid lysis function is more active, such as hypercoagulable conditions, disseminated intravascular coagulation function, kidney disease, liver transplant rejection, thrombolytic therapy, etc. As long as there is active thrombosis and fibrinolytic activity in the body's blood vessels, D-dimer will immediately rise. Myocardial infarction, cerebral infarction, pulmonary embolism, venous thrombosis, surgical treatment, tumors, focal intravascular coagulation, infection and tissue necrosis can all cause an increase in D-dimer. This is especially true for the elderly and hospitalized patients, as diseases such as bacteremia can easily cause abnormal coagulation function and lead to an increase in D-dimer. |
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