Who is prone to polycystic ovary

Who is prone to polycystic ovary

When many girls are diagnosed with polycystic ovary disease, they are very worried, because patients worry that if the treatment or plastic surgery fails, the condition will become more serious, and they are also worried that they don’t know how the disease will worsen. So who is prone to polycystic ovary disease? Let’s take a look at this issue together below.

Who is most likely to develop polycystic ovary disease?

1. Occurred during marriage and childbearing, about 85% of them are between 22 and 31 years old.

2. Those with infrequent menstruation, scanty menstruation, secondary amenorrhea, anovulatory menstruation, menorrhagia, frequent or multifunctional abnormal uterine bleeding.

3. Infertility, with primary infertility being the most common.

4. Excessive hair growth and obesity are most obvious on the upper lips, arms, and lower limbs. There will be one to multiple roots of hair around the nipples and the center line of the lower abdomen. Hair distribution has masculine characteristics. Or there may be mere weight gain without significant obesity.

5. During gynecological examination, about 67% of patients can feel the uterus and ovaries on one or both sides.

Who is prone to polycystic ovary disease? The above introduction is summarized here for everyone. I hope that women with polycystic ovary disease can seriously treat their polycystic ovary disease symptoms and don’t worry about treatment. If the hospital you choose is a professional hospital, there will not be any risks in the treatment of polycystic ovary disease, and it is hoped that the patient will have some psychological adjustments.

After suffering from polycystic ovary disease, the patient will first have problems with menstruation, and in some cases, anovulation may occur. Some severe patients may even experience premature menopause or a significant decrease in menstrual flow.

At this time, timely treatment is needed, otherwise the patient's uterine wall will be stimulated by simple high androgen for a long time, resulting in atypical hyperplasia of the uterine wall and endometrial cancer.

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