What to do if endometrial thickening and bleeding do not stop

Endometrial hyperplasia is the proliferation of uterine wall glandular ducts or interstitial cells due to the increase of endogenous or exogenous estrogen. The clinical manifestation is multifunctional abnormal uterine bleeding. It can occur in women of childbearing age and menopausal women. Endometrial hyperplasia, intestinal metaplasia and endometrial carcinoma are a continuous evolutionary process both in histology and molecular biology.

The thickness of the uterine wall is normal

1. Proliferative stage: The proliferative stage of women is generally from the fifth to the ninth day of menarche. At this time, the uterine wall of women is very thin. In the early stage of endometrial hyperplasia, the thickness of the endometrium is about 4-6mm. When it reaches the middle of the proliferative area, the thickness of the endometrium will increase to 8-1b250m. At the end of the proliferative stage, the thickness of the endometrium will continue to increase to about 9-1b250m.

2. Metabolism period: Generally within 1-5 days after the ovulation day of girls, the uterine wall of female friends will continue to thicken. Within 6-10 days after the ovulation period, the metabolism of the uterine wall of female friends will show a significant improvement. At the end of the metabolic period, the uterine wall of female friends is generally around 5-6mm.

3. Menstrual period: Within 1-4 days of the first menstruation, the female hormones in women's bodies will decrease to a certain extent, and the uterine wall will shed to a certain extent. The shed necrotic cells will be discharged along with menstruation. At this time, the thickness of women's endometrium is about 8-1b250m.

Standard of care

The treatment of intestinal metaplasia of the uterine wall must first establish a clear diagnosis and find out the cause of intestinal metaplasia, whether there is polycystic ovary, ovarian tumors or other endocrine and neurological disorders. Those with any of the above conditions require targeted treatment. At the same time, drug treatment can be started for intestinal metaplasia of the uterine wall, using drug treatment or surgical treatment.

The choice of these two treatment options should be based on age, type of endometrial hyperplasia, requirements for pregnancy, etc., and different solutions should be provided. It can be combined with some Chinese medicinal materials that can prevent lesions. Scientific research has confirmed that ginsenoside rh2 can inhibit cancer cell metastasis and induce tumor cell apoptosis.